duced by deletion of Anp32e, specifically in thymocytes. To a lot more closely examine this possible induction, we tested no matter if protein levels of Anp32a and Anp32b have been also elevated in Anp32e2/2 thymocytes. As shown in from serum starvation-induced arrest. These information suggest that Anp32e does not play a exclusive role in proliferation beneath either typical or low serum conditio