duced by deletion of Anp32e, especially in thymocytes. To extra closely examine this potential induction, we tested whether or not protein levels of Anp32a and Anp32b had been also improved in Anp32e2/2 thymocytes. As shown in from serum starvation-induced arrest. These information suggest that Anp32e will not play a distinctive role in proliferation beneath either regular or low serum circumstanc